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DCフィールド | 値 | 言語 |
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dc.contributor.author | 奥山, 明彦 | ja |
dc.contributor.author | 野々村, 祝夫 | ja |
dc.contributor.author | 中村, 正広 | ja |
dc.contributor.author | 並木, 幹夫 | ja |
dc.contributor.author | 松田, 稔 | ja |
dc.contributor.author | 土井, 康裕 | ja |
dc.contributor.author | 松井, 孝之 | ja |
dc.contributor.author | 高, 栄哲 | ja |
dc.contributor.author | 近藤, 宣幸 | ja |
dc.contributor.author | 竹山, 政美 | ja |
dc.contributor.author | 藤岡, 秀樹 | ja |
dc.contributor.alternative | OKUYAMA, Akihiko | en |
dc.contributor.alternative | NONOMURA, Norio | en |
dc.contributor.alternative | NAKAMURA, Masahiro | en |
dc.contributor.alternative | NAMIKI, Mikio | en |
dc.contributor.alternative | MATSUDA, Minoru | en |
dc.contributor.alternative | DOI, Yasuhiro | en |
dc.contributor.alternative | MATSUI, Takayuki | en |
dc.contributor.alternative | KOH, Eitetsu | en |
dc.contributor.alternative | KONDOH, Nobuyuki | en |
dc.contributor.alternative | TAKEYAMA, Masami | en |
dc.contributor.alternative | FUJIOKA, Hideki | en |
dc.date.accessioned | 2010-06-02T02:58:25Z | - |
dc.date.available | 2010-06-02T02:58:25Z | - |
dc.date.issued | 1988-11 | - |
dc.identifier.issn | 0018-1994 | - |
dc.identifier.uri | http://hdl.handle.net/2433/119773 | - |
dc.description.abstract | In vivo and in vitro studies were performed to determine the function of Leydig and Sertoli cells of the human testis with various degrees of spermatogenic impairment. The increases in basal and peak serum levels of luteinizing hormone (LH) and follicle stimulating hormone (FSH) after LH-RH administration correlated with the degree of impairment of spermatogenesis, and the basal peripheral blood levels of testosterone and that after human chorionic gonadotropin (hCG) administration for patients with moderate or severe impairment were significantly lower than the values for those with mild impairment. The concentration of testosterone in the internal spermatic vein of varicocele patients with or without hCG treatment did not differ between in mild and moderate impairment. In studies on cultured Sertoli cells, the production rate of plasminogen activator in patients with severe impairment was significantly lower than that in patients with moderate or mild impairment. The decrease in testicular high-affinity binding site for FSH correlated with the degree of hypospermatogenesis found in idiopathic male infertility, but, on the contrary, the hCG (LH) receptors showed no correlation with the degree of impairment of spermatogenesis. In the investigation of the relationship between testicular FSH receptors and the effectiveness of human menopausal gonadotropin (hMG)-hCG treatment on idiopathic male infertility, the presence or absence of testicular FSH receptors predicted the responsiveness to the treatment. | en |
dc.format.mimetype | application/pdf | - |
dc.language.iso | jpn | - |
dc.publisher | 泌尿器科紀要刊行会 | ja |
dc.subject | Spermatogenic impairment | en |
dc.subject | Endocrinology | en |
dc.subject | Leydig cell | en |
dc.subject | Sertoli cell | en |
dc.subject.ndc | 494.9 | - |
dc.title | 精子形成障害の内分泌学的背景 - ライディヒおよびセルトリ細胞機能のin vivoおよびin vitroでの評価 - | ja |
dc.title.alternative | Endocrinological profile of human spermatogenic impairment--evaluation of function of Leydig and Sertoli cells in vivo or in vitro studies | en |
dc.type | departmental bulletin paper | - |
dc.type.niitype | Departmental Bulletin Paper | - |
dc.identifier.ncid | AN00208315 | - |
dc.identifier.jtitle | 泌尿器科紀要 | ja |
dc.identifier.volume | 34 | - |
dc.identifier.issue | 11 | - |
dc.identifier.spage | 1943 | - |
dc.identifier.epage | 1948 | - |
dc.textversion | publisher | - |
dc.sortkey | 14 | - |
dc.address | 大阪大学医学部泌尿器科学教室 | ja |
dc.address | 大阪大学医学部泌尿器科学教室 | ja |
dc.address | 大阪大学医学部泌尿器科学教室 | ja |
dc.address | 大阪大学医学部泌尿器科学教室 | ja |
dc.address | 大阪大学医学部泌尿器科学教室 | ja |
dc.address | 健康保健組合連合会大阪中央病院泌尿器科 | ja |
dc.address | 健康保健組合連合会大阪中央病院泌尿器科 | ja |
dc.address | 健康保健組合連合会大阪中央病院泌尿器科 | ja |
dc.address | 健康保健組合連合会大阪中央病院泌尿器科 | ja |
dc.address | 健康保健組合連合会大阪中央病院泌尿器科 | ja |
dc.address | 健康保健組合連合会大阪中央病院泌尿器科 | ja |
dc.address.alternative | the Department of Urology, Osaka University Hospital | en |
dc.address.alternative | the Department of Urology, Osaka University Hospital | en |
dc.address.alternative | the Department of Urology, Osaka University Hospital | en |
dc.address.alternative | the Department of Urology, Osaka University Hospital | en |
dc.address.alternative | the Department of Urology, Osaka University Hospital | en |
dc.address.alternative | the Department of Urology, Kenkohoken-kumiai.rengoka Oisaka- Central Hospital | en |
dc.address.alternative | the Department of Urology, Kenkohoken-kumiai.rengoka Oisaka- Central Hospital | en |
dc.address.alternative | the Department of Urology, Kenkohoken-kumiai.rengoka Oisaka- Central Hospital | en |
dc.address.alternative | the Department of Urology, Kenkohoken-kumiai.rengoka Oisaka- Central Hospital | en |
dc.address.alternative | the Department of Urology, Kenkohoken-kumiai.rengoka Oisaka- Central Hospital | en |
dc.address.alternative | the Department of Urology, Kenkohoken-kumiai.rengoka Oisaka- Central Hospital | en |
dc.identifier.pmid | 3149448 | - |
dcterms.accessRights | open access | - |
dc.identifier.pissn | 0018-1994 | - |
dc.identifier.jtitle-alternative | Acta urologica Japonica | la |
dc.identifier.jtitle-alternative | Hinyokika Kiyo | en |
出現コレクション: | Vol.34 No.11 |
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