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タイトル: Project 4 Project Research on the Elucidation of Generating Mechanism of Damaged Protein Induced by Aging and Irradiation (27P4)
著者: Fujii, N.
発行日: Aug-2016
出版者: The Research Reactor Institute, Kyoto University
誌名: KURRI Progress Report
巻: 2015
号: APRIL 2015 – MARCH 2016
開始ページ: 16
終了ページ: 23
目次: PR4-1 Detection and Tissue Distribution of D-Aspartyl Endopeptidases Activity in the Ascidian (Ciona intestinalis). T. Kinouchi and N. Fujii (27P4-1) [17]
PR4-2 Damage to Biological Molecules Induced by Ionizing Irradiation and Biological Defense Mechanisms against Ionizing Radiation II. T. Saito et al. (27P4-2) [18]
PR4-3 Separation Condition of the Peptides Containing Isomerized Asp Residues after Treatment of Protein L-Isoaspartyl Methyltransferase (PIMT). Y. Sadakane and N. Fujii (27P4-3) [19]
PR4-4 Analysis of Environmental Stress-related Hearing Loss in Mice and Humans. N. Ohgami and N.Fujii (27P4-4) [20]
PR4-5 Identification of Biologically Uncommon β-aspratyl Residues in Proteins using LC-MS/MS. N. Fujii et al. (27P4-5) [21]
PR4-6 D-β-Asp Containing Peptides are Resistant to Peptide Bond Cleavage Compared with L-α-Asp Containing Peptides. K. Aki and E. Okamura (27P4-6) [22]
PR4-7 One-shot LC-MS/MS Analysis of Deamidation of Rat Lens α- and β-crystallins Induced by γ-irradiation. N. Fujii et al. (27P4-7) [23]
URI: http://hdl.handle.net/2433/218560
関連リンク: http://www.rri.kyoto-u.ac.jp/PUB/report/PR/ProgRep2015/ProgRep2015.html
出現コレクション:2015

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