ダウンロード数: 209
このアイテムのファイル:
| ファイル | 記述 | サイズ | フォーマット | |
|---|---|---|---|---|
| KURRI_PR2016_3.pdf | 1.18 MB | Adobe PDF | 見る/開く |
| タイトル: | Project 2 Project Research on the Elucidation of Generating Mechanism of Damaged Protein Induced by Aging and Irradiation (28P2) |
| 著者: | Fujii, N. |
| 発行日: | Jul-2017 |
| 出版者: | The Research Reactor Institute, Kyoto University |
| 誌名: | KURRI Progress Report |
| 巻: | 2016 |
| 号: | APRIL 2016 - MARCH 2017 |
| 開始ページ: | 3 |
| 終了ページ: | 10 |
| 目次: | PR2-1 Consideration for the Distribution of D-Aspartyl Endopeptidases Activity in Various Living Things /T. Kinouchi and N. Fujii (28P2-1) [4] PR2-2 Damage to Biological Molecules Induced by Ionizing Irradiation and Biological Defense Mechanisms against Ionizing Radiation III /T. Saito and N. Fujii (28P2-2) [5] PR2-3 Separation Condition of the Prion Peptide (106-126) after Treatment of Protein L-Isoaspartyl Methyltransferase (PIMT) /Y. Sadakane and N. Fujii (28P2-3) [6] PR2-4 Analysis of Hearing Impairments in Mice Exposed to Environmental Stress /N. Ohgami and N. Fujii (28P2-4) [7] PR2-5 Identification of Contiguous β-aspratyl Residues in Peptide Using MS /N. Fujii et al. (28P2-5) [8] PR2-6 The Stability of D-β-Asp :kinetics of the Competitive Reactions of Isomerization and Peptide bond Cleavage /K. Aki et al. (28P2-6) [9] PR2-7 Change of Protein Function by the Replacement of a Single Aspartyl Isomer in a Protein /N. Fujii et al. (28P2-7) [10] |
| 記述: | In case that corrections are made, an errata will be provided in the following webpage: http://www.rri.kyoto-u.ac.jp/PUB/report/PR/ProgRep2016/ProgRep2016.html |
| URI: | http://hdl.handle.net/2433/227270 |
| 関連リンク: | http://www.rri.kyoto-u.ac.jp/PUB/report/PR/ProgRep2016/ProgRep2016.html |
| 出現コレクション: | 2016 |
このリポジトリに保管されているアイテムはすべて著作権により保護されています。