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Title: The source-filter theory of whistle-like calls in marmosets: Acoustic analysis and simulation of helium-modulated voices.
Authors: Koda, Hiroki  kyouindb  KAKEN_id
Tokuda, Isao T
Wakita, Masumi  kyouindb  KAKEN_id
Ito, Tsuyoshi  kyouindb  KAKEN_id  orcid (unconfirmed)
Nishimura, Takeshi  kyouindb  KAKEN_id  orcid (unconfirmed)
Author's alias: 西村, 剛
Issue Date: Jun-2015
Publisher: AIP Publishing
Journal title: The Journal of the Acoustical Society of America
Volume: 137
Issue: 6
Start page: 3068
End page: 3076
Abstract: Whistle-like high-pitched "phee" calls are often used as long-distance vocal advertisements by small-bodied marmosets and tamarins in the dense forests of South America. While the source-filter theory proposes that vibration of the vocal fold is modified independently from the resonance of the supralaryngeal vocal tract (SVT) in human speech, a source-filter coupling that constrains the vibration frequency to SVT resonance effectively produces loud tonal sounds in some musical instruments. Here, a combined approach of acoustic analyses and simulation with helium-modulated voices was used to show that phee calls are produced principally with the same mechanism as in human speech. The animal keeps the fundamental frequency (f0) close to the first formant (F1) of the SVT, to amplify f0. Although f0 and F1 are primarily independent, the degree of their tuning can be strengthened further by a flexible source-filter interaction, the variable strength of which depends upon the cross-sectional area of the laryngeal cavity. The results highlight the evolutionary antiquity and universality of the source-filter model in primates, but the study can also explore the diversification of vocal physiology, including source-filter interaction and its anatomical basis in non-human primates.
Rights: © 2015 Acoustical Society of America. This article may be downloaded for personal use only. Any other use requires prior permission of the author and the American Institute of Physics.
DOI(Published Version): 10.1121/1.4921607
PubMed ID: 26093398
Appears in Collections:Journal Articles

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